Abstract Archives of the RSNA, 2014
GIE209
Don’t Be Afraid of the Dark: A Practical Approach to T2- Hypointense Focal Liver Lesions
Education Exhibits
Presented in 2014
Bianca Guedes Ribeiro MD, Presenter: Nothing to Disclose
Romulo Varella MD, Abstract Co-Author: Nothing to Disclose
Natalia Sabaneeff MD, Abstract Co-Author: Nothing to Disclose
Augusto Guimaraes Altoe, Abstract Co-Author: Nothing to Disclose
Leonardo Kayat Bittencourt MD, MSc, Abstract Co-Author: Nothing to Disclose
Vitor Moreira Sardenberg MD, Abstract Co-Author: Nothing to Disclose
Focal liver lesions are common findings in magnetic resonance imaging ( MRI) and the vast majority of them manifest as hyperintense on T2-weighted images .
However, hypointense T2 images , although less common , can be found in numerous pathologies, and this findins may help to narrow the list of differential diagnoses.
Focal liver lesions (both benign and malignant) with low signal intensity on T2-WI include: iron deposition, calcifications, blood products, necrosis / necrotic nodules and lesions containing mucin.
Each topic will be discussed and illustrated below :
1 - Review of protocols used in MRI for focal liver lesions
2 - Main causes of lesions with low signal intensity on T2WI
- Iron deposition
- Calcifications (residual granuloma)
- Degradation products of blood
- Necrosis
- Injuries containing mucin
3 - Common lesions that may present with low signal intensity on T2WI:
- Focal nodular hyperplasia
- Hepatocellular adenoma
- Hepatocellular carcinoma
- Cholangiocarcinoma
- Metastases with and without mucin
- Dysplastic / regenerative nodules
- Hydatid cyst
- Epithelioid hemangioendothelioma
http://abstract.rsna.org/uploads/2014/14015201/14015201_wth3.pdf
Guedes Ribeiro, B,
Varella, R,
Sabaneeff, N,
Altoe, A,
Bittencourt, L,
Sardenberg, V,
Don’t Be Afraid of the Dark: A Practical Approach to T2- Hypointense Focal Liver Lesions. Radiological Society of North America 2014 Scientific Assembly and Annual Meeting, - ,Chicago IL.
http://archive.rsna.org/2014/14015201.html